DermKnowledgeBASE: Seborrheic

Seborrheic

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The text is the summary of recent articles on Seborrheic at 75 thresold from National Library of Medicine (NLM). This information is subject to NCBI's Disclaimer and Copyright notice.


Various environmental and intrinsic factors have been identified as predisposing factors for SD, but its etiology remains poorly understood [1]. The clinical manifestations of DD are characterized by Warty Papules and Plaques in seborrheic areas, and association with neuropsychiatric abnormalities has also been reported in a few families with DD [2]. Clinicians are frequently presented with a challenge when determining whether to remove a seborrheic keratosis, and which treatment modality to use when doing so [3]. We sought to analyze dermoscopy use in US Dermatology residencies to better understand resident dermoscopy utilization and teaching modalities [4]. We found residents learn dermoscopy via Multiple teaching modalities [5].

The most commonly reported dermoscopy teaching modality was didactic lectures, followed by time in clinic with a dermoscopy expert [6]. Of the different teaching modalities, time in the clinic with a dermoscopy expert was reported to be the most effective [7]. Narrow-band ultraviolet-B phototherapy appears to be a very effective and safe treatment option for patients with severe seborrheic dermatitis [8]. To the best of our knowledge, there has been no previous study describing dermatologic manifestations in Mauritanians infected with HIV [9]. There are different Malassezia species, which have been recently reported to be resistant to common antifungals [10].

They have been associated with dermatological diseases such as seborrheic Dermatitis, pityriasis versicolor, atopic dermatitis, and folliculitis [11]. Not all conditions require immediate dermatological treatment and can be managed by targeted skin care interventions [12]. The authors present a case of a 69-year-old man with a left shoulder lesion that displayed characteristic clinical and microscopic features of seborrheic Keratosis on Biopsy [13]. These stained strongly with Periodic acid-Schiff and were diastase sensitive, suggestive of glycogen accumulation and possible trichilemmal differentiation [14]. It is clinically manifested by Hyperkeratotic Papules primarily affecting seborrheic areas on the head, Neck and thorax, with less frequent involvement of the Oral Mucosa [15].

Important differences in the presentation of common dermatoses such as seborrheic dermatitis and acne exist in patients with darker skin types [16]. Such treatments may result in adverse effects such as postinflammatory hyperpigmentation or keloid scarring at a higher rate [17]. These lesions also exhibit different demographics, with dermatosis papulosa nigra having a predilection for dark-skinned individuals and a Female predominance [18]. No studies to date have investigated this, but studies assessing the mechanisms of similar dermatologic conditions may yield significant clues [19]. In fact eccrine poroma in the postauricular area has only been rarely reported [20].

Histological examination showed distinct features, and eccrine poroma was diagnosed [21]. The frequency of eccrine poroma is dependent on eccrine sweat glands density, and thus, usually occurs on the palms or Soles [22]. For eccrine poroma in the Head and Neck region, the differential diagnosis must rule out other masses, such as nevus, skin tag, pyogenic granuloma, Cyst, basal cell carcinoma, and seborrheic keratosis [23]. Some lesions of BD are often difficult to be differentiated from seborrheic Keratosis, actinic keratosis, lichen planus-like keratosis, etc [24]. NICF is characterized by Multiple folliculocentric Papules with a predilection for occurring in seborrheic areas in adults and corresponding dilated follicular ostia containing crystalline material [25].

The precise pathogenesis and nature of this crystalline material are currently unknown [26]. In addition, we present analysis using infrared Microscopy for improved characterization of this crystalline material [27]. At T1, a complete clinical and dermoscopic resolution was observed in 37 lesions after an average of 3 applications/lesion (range 2-4) [28]. The histopathological study demonstrated a seborrheic keratosis in the peripheral areas, whereas the central part of the Lesion was a sebaceoma [29]. These, together with the presence of Hair, provide a suitable environment for superficial infections, infestations and Inflammatory diseases [30].

The disease, despite having clinical features similar to Dermatitis, does not have the same histopathologic features or the same Progressive clinical behavior [31]. They are usually inflammatory in nature and may be encountered as Leser-Trélat Sign [32]. This review discusses the recent findings in dandruff and seborrheic dermatitis and their relation to the skin microbiota [33]. A total of 226 patients (205 females and 21 males) with suspected Scalp contact Dermatitis were identified, and the Patch test results and clinical data for those patients were analyzed [34].

References: 1 , 2 , 3 , 4 , 5 , 6 , 7 , 8 , 9 , 10 , 11 , 12 , 13 , 14 , 15 , 16 , 17 , 18 , 19 , 20 , 21 , 22 , 23 , 24 , 25 , 26 , 27 , 28 , 29 , 30 , 31 , 32 , 33 , 34 ,

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