DermKnowledgeBASE: Mucinosis Follicularis

Mucinosis Follicularis

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The text is the summary of recent articles on Mucinosis Follicularis at 75 thresold from National Library of Medicine (NLM). This information is subject to NCBI's Disclaimer and Copyright notice.


Histologically, cicatricial alopecia is characterized by Dermal scarring, along with absent or reduced Hair follicles and reduced Number of erector pili muscles [1]. Clinical evaluation was compared with histopathologic analysis of follicular architecture, as well as with the type, localization and extent of Inflammatory Infiltrate [2]. Our experience indicates the need of more complex research to extend the knowledge about the etiopathogenesis and treatment options for cicatricial alopecia [3]. The progressive course of the disease and the unresponsiveness to treatments support the current view that SLHA is a syringotropic variant of mucinosis follicularis and therefore should be viewed as a facultative precursor Lesion of mycosis fungoides [4]. In our patient, during a 7-year follow-up, no T-cell lymphoma occurred [5].

It is normally differentiated into a Juvenile Benign form and an Adult form possibly associated with mycosis fungoides [6]. Three months later, he developed Erythematous Patches and Plaques on his whole body, accompanied by cervical lymphadenopathy [7]. Histologically, the strong pathological changes began within the sebaceous glands and secondarily occurred in other parts of the pilosebaceous unit [8]. We could demonstrate by electron Microscopy that all the follicular cells in this process were undifferentiated Sebaceous Cells [9]. Provocation with standardized light testing was carried out on both normally Pigmented skin and on areas of Hypopigmentation representing a residual state after earlier skin lesions but without any signs of active mucinosis follicularis [10].

References: 1 , 2 , 3 , 4 , 5 , 6 , 7 , 8 , 9 , 10 ,

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