DermKnowledgeBASE: Hamartoma

Hamartoma

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The text is the summary of recent articles on Hamartoma at 75 thresold from National Library of Medicine (NLM). This information is subject to NCBI's Disclaimer and Copyright notice.


All cases exhibited intraparenchymal proliferation of variable size, thick-walled blood vessels within collagenous stroma containing a Sparse to focally cellular population of haphazardly distributed smooth Muscle Cells [1]. The Lesion exhibited low density on plain computed tomography and no enhancement in 4 cases, and inhomogeneous enhancement in 1 case [2]. An SMH can also rarely originate from smooth muscles of vessels in palmoplantar skin with the absence of pilosebaceous units [3]. Normal intestinal epithelial NCM460 Cells were transfected with LKB1 shRNA plasmid or negative control via lentiviral vectors, and the role of LKB1 in cell polarization and migration were determined using CCK8 and Transwell assays [4]. Among survivors, three had early complications and eight had late complications [5].

Early complications included anastomotic leak (2) and lung compression by stomach (1) [6]. If the Lesion grows to a large size, a series of clinical manifestations and serious complications may occur [7]. Postoperative histopathological examination confirmed the Lesion as a giant Renal AML [8]. Skeletal lesions are asymptomatic and are detected by means of iterative X-ray [9].

References: 1 , 2 , 3 , 4 , 5 , 6 , 7 , 8 , 9 ,

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